– 150 –
80 GS/FR – PARIS, May 2012
RESOLUTION No. 16
Recognition of the Bovine Spongiform Encephalopathy Risk Status of Member
Countries CONSIDERING THAT
1. During the 67th General Session the OIE World Assembly of Delegates
(Assembly) established a procedure for annually updating a list of Member
Countries, categorised by their bovine spongiform encephalopathy (BSE) risk
according to the provisions of the Terrestrial Animal Health Code (Terrestrial
Code),
2. During the 76th General Session, the Assembly adopted Resolution No.
XXII, which specified and updated the procedure for Member Countries to follow
to achieve official recognition and maintenance of status of certain
diseases,
3. During the 76th General Session, the Assembly adopted Resolution No.
XXIII, which specified the financial implications for Member Countries applying
for evaluation of official recognition or re-instatement of a BSE risk status to
meet part of the costs defrayed by the OIE in the evaluation process,
4. Information published by the OIE is derived from declarations made by
the OIE Delegate of Member Countries. The OIE is not responsible for publication
and maintenance of Member Countries disease status based on inaccurate
information or non-reporting of changes in epidemiological status or other
significant events subsequent to the time of declaration of the BSE risk
status.
THE ASSEMBLY
RESOLVES THAT
1. The Director General publish the following list of Member Countries
recognised as having a negligible BSE risk in accordance with Chapter 11.5. of
the Terrestrial Code:
Argentina
Australia
Austria
Belgium
Brazil
Chile
Colombia
Denmark
Finland
Iceland
India
New Zealand
Norway
Panama
Paraguay
Peru
Singapore
Sweden
Uruguay
– 151 –
80 GS/FR – PARIS, May 2012
2. The Director General publish the following list of Member Countries
recognised as having a controlled BSE risk in accordance with Chapter 11.5. of
the Terrestrial Code:
Canada
Chinese Taipei
Croatia
Cyprus
Czech Republic
Estonia
France
Germany
Greece
Hungary
Ireland
Italy
Japan
Korea (Rep. of)
Latvia
Lichtenstein
Lithuania
Luxembourg
Malta
Mexico
Netherlands
Nicaragua
Poland
Portugal
Slovak Republic
Slovenia
Spain
Switzerland
United Kingdom
United States of America
AND
3. The Delegates of these Member Countries shall immediately notify the
Headquarters if BSE occurs in their countries or their territories.
_______________
(Adopted by the World Assembly of Delegates of the OIE on 22 May 2012)
The Delegate of Denmark, speaking on behalf of the 27 EU Member States,
thanked Dr Thiermann and the Code Commission for the excellent work and
supported the proposed work programme. The Delegate again requested the drafting
of an introductory chapter to the Code, setting out which recommendations of the
Code were relevant to international trade. The Delegate also recommended a
partial revision of Chapter 14.9. (Scrapie), based on the written justification
provided by the EU prior to this General Session.
328. Agent causing chronic wasting disease (CWD)
Dr Ben Jebara summarised the situation of the agent causing chronic wasting
disease (CWD) which was a transmissible spongiform encephalopathy (TSE), along
with other spongiform diseases, such as scrapie and bovine spongiform
encephalopathy. At the present time there was no scientific evidence that the
infection was transmissible to domestic animals or to humans. Two countries
reported the disease present in 2011: United States of America and Canada.
– 104 –
80 GS/FR – PARIS, May 2012
United States of America
According to the information provided by the APHIS CWD website (official
USA government website) the species known to be susceptible to CWD in North
America were elk (Cervus canadensis), mule deer (Odocoileus hemionus), Columbian
black-tailed deer (Odocoileus hemionus columbianus), white-tailed deer
(Odocoileus virginianus) and, possibly the red deer (Cervus elaphus) due to its
genetic similarity to elk.
APHIS reported that the disease had been identified in different States in
wild deer, moose and elk (Colorado, Illinois, Kansas, Minnesota, Missouri,
Nebraska, New Mexico, New York, North Dakota, South Dakota, Utah, Virginia, West
Virginia, Wisconsin, Wyoming) and in farmed elk and deer herds (Colorado,
Kansas, Minnesota, Montana, Nebraska, Oklahoma, South Dakota and
Wisconsin).
According to the Questionnaire on Wildlife Diseases for 2011, the presence
of the disease in the United States of America was limited to various zones. A
total of 20,430 farmed elks were tested for surveillance purposes and two new
elk herds were found to be CWD positive – each with at least one CWD-positive
elk. During the past 10 years, CWD has been detected in 52 farmed herds (39 elk
herds and 13 white-tailed deer herds) in 11 States in the United States of
America. Data resulting from 2011 sampling would be available in late
2012.
Canada
According to the Questionnaire on Wildlife Diseases for 2011, the
occurrence of infection (without clinical signs) in Canada was limited to
various zones. Out of a total of 54 cases reported in wild animals, 45 were in
mule deer (Odocoileus hemionus), eight in white-tailed deer (Odocoileus
virginianus) and one in an elk (Cervus canadensis). The disease has been
identified in two Provinces, Saskatchewan and Alberta.
Final Report of the 80th General Session, 20 - 25 May 2012 (pdf file,
4021Kb)
http://www.oie.int/about-us/final-reports-of-the-general-session-of-the-oie-international-committee/
2002
Subject: Re: CWD AMERICA ???
Date: Fri, 12 Jul 2002 19:10:18 +0200
From: "INFORMATION DEPT" Organization: O.I.E
To: "Terry S. Singeltary Sr."
References:
I agree with you Dr Terry. The OIE, namely the International Animal Health Code Commission is working on making proposals to Member Countries to change the OIE lists so to avoid some the problems mentioned in you e-mail. This will take at least two years before adoption by the International Committee. For BSE, countries asked the OIE to post information on BSE on the OIE web site.
Personally, I am interested in Chronic Wasting Disease and I follow what is distributed through ProMed. Delegates of OIE Member Countries can propose diseases to be added to the list.
Kind regards.
Karim Ben Jebara
Subject: Re: CWD AMERICA ???
Date: Fri, 12 Jul 2002 19:10:18 +0200
From: "INFORMATION DEPT" Organization: O.I.E
To: "Terry S. Singeltary Sr."
References:
I agree with you Dr Terry. The OIE, namely the International Animal Health Code Commission is working on making proposals to Member Countries to change the OIE lists so to avoid some the problems mentioned in you e-mail. This will take at least two years before adoption by the International Committee. For BSE, countries asked the OIE to post information on BSE on the OIE web site.
Personally, I am interested in Chronic Wasting Disease and I follow what is distributed through ProMed. Delegates of OIE Member Countries can propose diseases to be added to the list.
Kind regards.
Karim Ben Jebara
=========================================
----- Original Message -----
From: "Terry S. Singeltary Sr."
To: "INFORMATION DEPT"
Sent: Friday, July 12, 2002 8:43 PM
Subject: Re: CWD AMERICA ???
hello Dr. Jebara,
many thanks for your swift and kind reply.
if i am not mistaken, it was the same email address. it was 3 or 4 weeks ago i wrote, as it is, i don't save 'sent' emails anymore, unless very important.
my main concern (besides the fact that a potential TSE has been in the USA cattle for some time, but the APHIS do not test to find), is that the CWD could very well be transmitting to humans, and i just did not see to much posted about it on OIE site.
Coming back to your question, Chronic Wasting Disease is not an OIE
listed disease. Please see OIE disease lists at
http://www.oie.int/eng/maladies/en_classification.htm#ListeA).
why is this TSE (CWD) not listed and followed as with BSE ?'
why is this TSE (CWD) not listed and followed as with BSE ?'
Article 1.1.3.2. 1. Countries shall make available to other countries, through the OIE, whatever information is necessary to minimise the spread of important animal diseases and to assist in achieving better worldwide control of these diseases.
http://www.oie.int/eng/normes/MCode/A_00005.htm
The USA CWD is an important animal disease.
why is it not followed?
The decision to add or delete a disease from the OIE lists, come through proposals made by Member Countries and it has to be adopted by the International Committee.
i _urgently_ suggest a proposal to the OIE to follow this disease very closely, and to propose _more_ testing in the USA for TSEs in the USA cattle...
The USA CWD is an important animal disease.
why is it not followed?
The decision to add or delete a disease from the OIE lists, come through proposals made by Member Countries and it has to be adopted by the International Committee.
i _urgently_ suggest a proposal to the OIE to follow this disease very closely, and to propose _more_ testing in the USA for TSEs in the USA cattle...
kindest regards, terry
INFORMATION DEPT wrote:
Dear Sir,
This is the first time that I receive your e-mail. To whom have you written in the OIE or to which address?
Coming back to your question, Chronic Wasting Disease is not an OIE listed disease. Please see OIE disease lists at
http://www.oie.int/eng/maladies/en_classification.htm#ListeA).
Countries should report to the OIE any disease even is not listed in the OIE's lists in some conditions (example: an exceptional epidemiological event). Please read Chapter 1.1.3 of the International animal health code to have more information on disease notification and epidemiological information agreed by OIE Member Countries at : http://www.oie.int/eng/normes/MCode/A_00005.htm
The decision to add or delete a disease from the OIE lists, come through proposals made by Member Countries and it has to be adopted by the International Committee.
Countries should report to the OIE any disease even is not listed in the OIE's lists in some conditions (example: an exceptional epidemiological event). Please read Chapter 1.1.3 of the International animal health code to have more information on disease notification and epidemiological information agreed by OIE Member Countries at : http://www.oie.int/eng/normes/MCode/A_00005.htm
The decision to add or delete a disease from the OIE lists, come through proposals made by Member Countries and it has to be adopted by the International Committee.
Hope that I answered to your question.
Best regards.
Dr Karim Ben Jebara Head Animal Health Information Department OIE
----- Original Message -----
From: "Terry S. Singeltary Sr."
To:
Sent: Friday, July 12, 2002 6:18 PM
Subject: CWD AMERICA ???
I WROTE TO OIE RECENTLY ASKING 'WHY OIE DOES NOT FOLLOW CWD IN AMERICA' ? with no reply ? i am still seeking an answer ?
many thanks, and kind regards, terry
=====================
SNIP...
From: Terry S. Singeltary Sr.
To: wahis_devt@oie.int
Cc: m.zampaglione@oie.int ; oie@oie.int ; rma-mrr@tbs-sct.gc.ca ; B.Vallat@oie.int
Sent: Saturday, April 14, 2007 2:31 PM
Subject: TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES in the USA and OIE reporting of it ???
Greetings again OIE,
I am deeply concerned that the OIE has completely given up on the surveillance and eradication of TSE around the Globe. I am disappointed, and IF the OIE gives favorable ratings for the USA TSE rating with the new BSE/BASE MRR policy, I will then have lost all confidence of this organization as a regulatory authority on animal disease, and consider it nothing more than a National Trading Brokerage for all strains of animal TSE, just to satisfy there commodity. AS i said before, OIE should hang up there jock strap now, since it appears they will buckle every time a country makes some political hay about trade protocol, commodities and futures. IF they are not going to be science based, they should do everyone a favor and dissolve there organization. ...
1st and foremost question,
IF THE OIE gives favorable ratings for USA BSE/BASE/TSE, by what means will it be justified (scientific, not political) ??? ;
Sent: Sunday, January 28, 2007 9:12 PM
Subject: BSE; MRR; IMPORTATION OF LIVE BOVINES AND PRODUCTS DERIVED FROM BOVINES [Docket No. APHIS-2006-0041] RIN 0579-AC01 COMMENT SUBMISSION
January 28, 2007
Greetings APHIS,
I would kindly like to submit the following to ;
snip...
THE USA is in a most unique situation, one of unknown circumstances with human and animal TSE. THE USA has the most documented TSE in different species to date, with substrains growing in those species (BSE/BASE in cattle and CWD in deer and elk, there is evidence here with different strains), and we know that sheep scrapie has over 20 strains of the typical scrapie with atypical scrapie documented and also BSE is very likely to have passed to sheep. all of which have been rendered and fed back to animals for human and animal consumption, a frightening scenario. WE do not know the outcome, and to play with human life around the globe with the very likely TSE tainted products from the USA, in my opinion is like playing Russian roulette, of long duration, with potential long and enduring consequences, of which once done, cannot be undone. These are the facts as I have come to know through daily and extensive research of TSE over 9 years, since 12/14/97. I do not pretend to have all the answers, but i do know to continue to believe in the ukbsenvcjd only theory of transmission to humans of only this one strain from only this one TSE from only this one part of the globe, will only lead to further failures, and needless exposure to humans from all strains of TSE, and possibly many more needless deaths from TSE via a multitude of proven routes and sources via many studies with primates and rodents and other species.
MY personal belief, since you ask, is that not only the Canadian border, but the USA border, and the Mexican border should be sealed up tighter than a drum for exporting there TSE tainted products, until a validated, 100% sensitive test is available, and all animals for human and animal consumption are tested. all we are doing is the exact same thing the UK did with there mad cow poisoning when they exported it all over the globe, all the while knowing what they were doing. this BSE MRR policy is nothing more than a legal tool to do just exactly what the UK did, thanks to the OIE and GW, it's legal now. and they executed Saddam for poisoning ???
go figure. ...
Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518
Comment Submitted
Comment Receipt
Thank you. Your comment on Document ID: APHIS-2006-0041-0001 has been sent. Comment Tracking Number: APHIS-2006-0041-DRAFT-0028
Attachments:
C:\My Music\My Documents\APHIS-2006-0041_January 28.doc
If you wish to retain a copy of the receipt, use the following link to print a copy for your files.
http://www.regulations.gov/fdmspublic/component/main
SEE FULL TEXT OF MY SUBMISSION TO FEDERAL DOCKETS HERE ;
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0701&L=sanet-mg&T=0&P=3854
2nd question to OIE,
WHY HAS OIE FAILED TO REPORT THE NOR98 CASE DOCUMENTED IN THE USA ???
NOR98-LIKE STRAIN OF SCRAPIE FOUND IN WYOMING (1791 lines)
From: Terry S. Singeltary Sr. <[log in to unmask]>
Date: Wed, 11 Apr 2007 15:08:15 -0500
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=8315
AND, what about the DECLARATION OF EXTRAORDINARY EMERGENCY DUE TO ATYPICAL TSE IN THOSE MAD SHEEP OF MAD RIVER VALLEY ???
FOIA REQUEST FOR ATYPICAL TSE INFORMATION ON VERMONT SHEEP
Re: FOIA REQUEST FOR ATYPICAL TSE INFORMATION ON VERMONT SHEEP (4843 lines)
From: Terry S. Singeltary Sr. <[log in to unmask]>
Date: Mon, 2 Apr 2007 14:43:32 -0500
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=816
3RD question to OIE
,
WHY HAS OIE FAILED ON THERE PROMISE TO BRING THE REAL POTENTIAL FOR CWD RISK FACTORS TOWARD TRANSMISSION TO HUMANS TO THE ATTENTION OF THE PUBLIC AROUND THE GLOBE ???
Subject: Re: CWD AMERICA ???
Date: Fri, 12 Jul 2002 19:10:18 +0200
From: "INFORMATION DEPT"
Organization: O.I.E
To: "Terry S. Singeltary Sr."
References: <3D2F0169.3@wt.net> <012901c229b2$ad43bb90$7f00000a@HPKB>
3D2F2358.5010700@wt.net
I agree with you Dr Terry. The OIE, namely the International Animal
Health Code Commission is working on making proposals to Member
Countries to change the OIE lists so to avoid some the problems
mentioned in you e-mail. This will take at least two years before
adoption by the International Committee. For BSE, countries asked the
OIE to post information on BSE on the OIE web site.
Personally, I am interested in Chronic Wasting Disease and I follow what
is distributed through ProMed. Delegates of OIE Member Countries can
propose diseases to be added to the list.
Kind regards.
Karim Ben Jebara
----- Original Message -----
From: "Terry S. Singeltary Sr."
To: "INFORMATION DEPT"
Sent: Friday, July 12, 2002 8:43 PM
Subject: Re: CWD AMERICA ???
> hello Dr. Jebara,
>
> many thanks for your swift and kind reply.
>
> if i am not mistaken, it was the same email address.
> it was 3 or 4 weeks ago i wrote, as it is, i don't
> save 'sent' emails anymore, unless very important.
>
> my main concern (besides the fact that a potential TSE
> has been in the USA cattle for some time, but the APHIS
> do not test to find), is that the CWD could very well be
> transmitting to humans, and i just did not see to much
> posted about it on OIE site.
>
> > Coming back to your question, Chronic Wasting Disease is not an OIE
>
> > listed disease. Please see OIE disease lists at
>
> http://www.oie.int/eng/maladies/en_classification.htm#ListeA).
>
> why is this TSE (CWD) not listed and followed as with BSE ?
>
> Article 1.1.3.2.
> 1. Countries shall make available to other countries, through the
> OIE, whatever information is necessary to minimise the spread of
> important animal diseases and to assist in achieving better worldwide
> control of these diseases.
>
> http://www.oie.int/eng/normes/MCode/A_00005.htm
>
> The USA CWD is an important animal disease.
>
> why is it not followed?
>
> > The decision to add or delete a disease from the OIE lists, come
>
> > through proposals made by Member Countries and it has to be adopted by
>
> > the International Committee.
>
> i _urgently_ suggest a proposal to the OIE to follow this disease very
> closely, and to propose _more_ testing in the USA for TSEs in the USA
> cattle...
>
> kindest regards,
> terry
>
> INFORMATION DEPT wrote:
>
> > Dear Sir,
> >
> > This is the first time that I receive your e-mail. To whom have you
written
> > in the OIE or to which address?
> >
> > Coming back to your question, Chronic Wasting Disease is not an OIE
listed
> > disease. Please see OIE disease lists at
> > http://www.oie.int/eng/maladies/en_classification.htm#ListeA).
> >
> > Countries should report to the OIE any disease even is not listed
in the
> > OIE's lists in some conditions (example: an exceptional epidemiological
> > event). Please read Chapter 1.1.3 of the International animal health
code to
> > have more information on disease notification and epidemiological
> > information agreed by OIE Member Countries at :
> > http://www.oie.int/eng/normes/MCode/A_00005.htm
> >
> > The decision to add or delete a disease from the OIE lists, come
through
> > proposals made by Member Countries and it has to be adopted by the
> > International Committee.
> >
> > Hope that I answered to your question.
> >
> > Best regards.
> >
> > Dr Karim Ben Jebara
> > Head
> > Animal Health Information Department
> > OIE
> >
> >
> >
> > ----- Original Message -----
> > From: "Terry S. Singeltary Sr."
> > To:
> > Sent: Friday, July 12, 2002 6:18 PM
> > Subject: CWD AMERICA ???
> >
> >
> >
> >>I WROTE TO OIE RECENTLY ASKING 'WHY OIE DOES NOT FOLLOW CWD IN
> >>AMERICA' ? with no reply ? i am still seeking an answer ?
> >>
> >>many thanks,
> >>and kind regards,
> >>terry
=====================
SNIP...END
OIE needs to seriously consider making CWD (all strains) a Zoonotic Disease sooner, rather than later, after the fact, when millions have already become exposed.
why you ask, because CWD transmits to primates, as with BSE, and maybe humans as GSS ???
Re: Colorado Surveillance Program for Chronic Wasting Disease Transmission to Humans (TWO SUSPECT CASES) (8150 lines)
From: Terry S. Singeltary Sr. <[log in to unmask]>
Date: Wed, 4 Apr 2007 16:22:22 -0500
FURTHER into this case study, Colorado Surveillance Program for Chronic Wasting Disease Transmission to Humans
(TWO SUSPECT CASES) a look at case 1 and case 2 ;
CASE 1
A 52-year-old right-handed woman presented with a
1-year history of progressive memory loss, language impairment,
visuospatial disturbance, and myoclonus. She
related that she had been a histology technician in a laboratory
that processed tissue specimens from deer and elk
with CWD and had handled specimens without wearing
gloves. Both she and her family expressed significant
concerns about the possibility of transdermal transmission
of CWD. Her family history was negative for
dementia and other neurologic disorders. Brain magnetic
resonance imaging showed mild diffuse volume loss,
and electroencephalography demonstrated mild diffuse
slowing. Other laboratory studies were unremarkable. Cerebrospinal
fluid findings were unremarkable except for
a weakly immunostaining 14-3-3 protein band, an indeterminate
finding for the diagnosis of prion disease. Genetic
testing of the prion protein gene was normal, revealing
methionine homozygosity at codon 129. Brain
biopsy results were negative for the presence of proteaseresistant
prion protein but showed definite Alzheimer disease
with numerous neuritic plaques and tau-positive neurofibrillary
tangles (Figure). Further analysis of brain
tissue at the National Prion Disease Pathology Surveillance
Center was negative for prion disease by Western
blot analysis. Subsequent investigation by the state department
of health revealed the patient had worked in
an area of the laboratory that conducted necropsies on
domestic animals and had never been assigned to the
CWD testing laboratory. The Colorado Department of
Public Health and Environment could not confirm that
the technician had ever worked with deer and elk tissues.
CASE 2
This 25-year-old right-handed man had a 4-month history
of progressive gait disturbance, myoclonus, hallucinations,
slowed cognition, impaired attention, and
memory loss. He had hunted deer and elk in a CWD endemic
area of southern Wyoming and cooked and ate the
field-dressed meat. His family history was significant in
that his mother had died of a dementing disease at age
40 years, although there was neither a clinical diagnosis
nor an autopsy. Brain magnetic resonance imaging findings
were unremarkable, and electroencephalography
demonstrated 1-Hz high-amplitude periodic sharp wave
complexes. Other laboratory studies had negative results.
Testing for the 14-3-3 protein had positive results,
but the cerebrospinal fluid was otherwise unremarkable.
The diagnosis of Gerstmann-Stra¨ussler-Scheinker
syndrome, a familial prion disease, was confirmed with
a detailed autopsy examination and referral of the brain
to the National Prion Disease Pathology Surveillance Center.
Autopsy brain tissue showed the presence of proteaseresistant
prion protein by Western blot analysis. Genetic
evaluation revealed the P102L mutation in the prion protein
gene with methionine/valine heterozygosity at codon
129.
snip...end
I can't understand how they can keep claiming 'low, or no occupational transmission of CJD' ??? when there have been many cases that should have raised awareness, and in some cases they did, only to be swept under the rug as the infamous sporadic CJD, or some other TSE other than the nvCJD of the ukbsenvcjd only theory. it's a blown theory no one will accept too. lets look at a few occupational cases. ...TSS
now, some things to ponder ;
Questions:
1. Do neuritic plaques and tau-positive neurofibrillary tangles indicate
definite AD? Aren't these also found in GSS? What about concurrent AD
and TSE?
2. Are the NPDPSC results conclusive? Do WB results depend on the part
of the brain sampled?
3. Doesn't it seem unlikely the woman would flat-out lie about working
with CWD tissues? (I'm working on this locally.)
4. What about cross-contamination? The lab gets large numbers of
scrapie-infected sheep and CWD-infected deer and elk. I assume the
necropsy area is contaminated with TSEs.
snip...
>
> > Results Neuropathological and genetic assessment in the 2 patients proved
> the
>
> > diagnoses of early-onset Alzheimer disease and a rare genetic prion
> disease
>
>
> very interesting, and something to ponder here for sure ;
>
>
>
>
> AS implied in the Inset 25 we must not _ASSUME_ that
> transmission of BSE to other species will invariably
> present pathology typical of a scrapie-like disease.
>
> snip...
>
>
>
> http://www.bseinquiry.gov.uk/files/yb/1991/01/04004001.pdf
>
>
>
>
> and i think this would apply to CWD to humans as well.
>
>
>
>
> > rare genetic prion disease
>
>
>
>
> would be interesting to know the exact genetic TSE they are speaking of.
> GSS, FFI, Familial/Genetic CJD, and or the sporadic FFI that is not genetic,
> and don't ask me why ??? does not make sense to me either. it's either
> genetic or not. like i have said many times, the diagnostic criteria
> differentiating the different human and animal TSE is missing something. but
> if you have a strain of genetic/familial TSE i.e. FFI, and then you classify
> a sub-type of that strain that use to be gentic to sporadic, then you have
> either gone back to sCJD, or the complete damn diagnostic criteria is wrong.
> you just have well named the damn thing ;
>
>
>
>
> Parchi-Capellari-Chin-Schwarz-Schecter-Butts-Hudkins-Burns-Powers-Gambetti-D
> ISEASE.
>
> TSS
>
>
>
>
> Subject: Alzheimer-type neuropathology in a 28-year old patient with
> iatrogenic CJD after dural grafting
> Date: March 9, 2007 at 9:15 am PST
>
> HUMAN-04
snip...full text ;
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=1165
IN my opinion the WOAH/OIE is nothing more than a organized bunch of lobbyist for the members Countries in support of there INDUSTRY, bound together as one, with the only purpose of open trade for there precious commodities and futures. Speaking only of BSE, they failed at every corner, and then just said to hell with it, well just trade all strains of TSE globally.
snip...
NOW, ask yourself why not one single mad cow has been documented in the USA since the Honorable Phyllis Fong of the OIG did the end around Johanns, Dehaven et al ??? found two atypical BSE or BASE cases and they flat shut it down i tell you. IF the OIE gives a favorable rating, IF the OIE gives any other rating but the lowest, poorest possible BSE/TSE rating, the OIE will have sealed there fate once and for all, because most of the world knows the truth about the USA and there mad cows. THE OIE will then be able to stand side by side with the USA, and proudly claim to have sold there soul to the devil, all for a buck, commodities and futures, to hell with human health. A 'CONTROLLED' RATING IS EXACTLY what the OIE will get if that is what they classify the USA as a 'CONTROLLED RATING'. IT will be controlled by Johanns, Dehaven, and GW. IT WILL BE RIGGED in other words. but that is nothing new, it's been rigged for years. ...
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&D=0&P=498
Re: REPORT ON THE INVESTIGATION OF THE NINTH CASE OF BSE IN CANADA UPDATE MARCH 26, 2007 (921 lines)
From: Terry S. Singeltary Sr. <[log in to unmask]>
Date: Mon, 26 Mar 2007 15:48:11 -0600
Date: Tue, 10 Apr 2007 12:54:12 -0500
Reply-To: Sustainable Agriculture Network Discussion Group
<[log in to unmask]>
Sender: Sustainable Agriculture Network Discussion Group
<[log in to unmask]>
From: "Terry S. Singeltary Sr." <[log in to unmask]>
Subject: Re: Birth cohort of CANADIAN BSE-positive animal was exported to
the United States
Content-type: multipart/alternative;
Subject: Re: Birth cohort of CANADIAN BSE-positive animal was exported to the United States
Date: April 10, 2007 at 10:33 am PST
"It most likely" entered the food supply "given that it was slaughtered," said Karen Eggert, a spokeswoman with USDA's Animal and Plant Health Inspection Service.
"But it wouldn't have gone to slaughter if it was showing any clinical signs for BSE. We're not looking at this as a possibility that a BSE infected cow got into the United States," she said.
http://www.reuters.com/article/domesticNews/idUSN1040765520070410
how in the heck does she know ??? does she know what sub-clinical means ???
snip...full text ; http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0704&L=sanet-mg&T=0&P=7609
23.2 BSE-infected mad cows in the standing Canadian adult cattle population. very disturbing... http://www.aphis.usda.gov/newsroom/hot_issues/bse/downloads/BSE_Prevalence.pdf http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0703&L=sanet-mg&D=0&P=15653
BSE; MRR; IMPORTATION OF LIVE BOVINES AND PRODUCTS DERIVED FROM
BOVINES [Docket No. APHIS-2006-0041] RIN 0579-AC01
http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0701&L=sanet-mg&D=0&P=3854
NOW, FINAL QUESTION TO OIE, HOW CAN OIE JUSTIFY GIVING USA A FAVORABLE RATING ON BSE/BASE/TSE MRR POLICY WHEN THE USA HAS THE MOST DOCUMENTED TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHIES IN MORE SPECIES THAN ANY OTHER COUNTRY, ALL OF WHICH HAS BEEN RENDERED AND FED BACK TO ANIMALS (CATTLE INCLUDED) FOR HUMAN AND ANIMAL CONSUMPTION, AND ALSO HAS THE MOST DOCUMENTED ATTEMPTS AT COVERING UP MAD COW DISEASES IN THE USA, ALSO, MORE BLATANTLY AND HAPHAZARDLY THAN ANY OTHER COUNTRY IN THE WORLD, HOW CAN ONE JUSTIFY A FAVORABLE MAD COW RATING WITH ALL THIS $$$
THE ONLY BSE MRR RATING THE USA AND ALL OF NORTH AMERICA SHOULD GET IS A TERRIBLY FAILED RATING, SCIENTIFICALLY SPEAKING, THIS IS THE ONLY RATING POSSIBLE. ANY OTHER RATING WILL PROVE THE OIE IS NOTHING MORE THAN A FAILED AUTHORITY ON HUMAN/ANIMAL DISEASE, AND THEIR MOTO OF ''Protecting the world from emerging diseases linked to globalisation'' will read more like ''PROTECTING OUR COMMODITIES AND FUTURES FROM DEAD CONSUMERS FAMILIES LINKED TO EMERGING POLITICS''
I am still sincerely disgusted,
Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518
Wednesday, April 06, 2011
Presence and Seeding Activity of Pathological Prion Protein (PrPTSE) in Skeletal Muscles of White-Tailed Deer Infected with Chronic Wasting Disease
http://chronic-wasting-disease.blogspot.com/2011/04/presence-and-seeding-activity-of.html
CWD has been identified in free-ranging cervids in 15 US states and 2 Canadian provinces and in ≈ 100 captive herds in 15 states and provinces and in South Korea (Figure 1, panel B).
SNIP...
Long-term effects of CWD on cervid populations and ecosystems remain unclear as the disease continues to spread and prevalence increases. In captive herds, CWD might persist at high levels and lead to complete herd destruction in the absence of human culling. Epidemiologic modeling suggests the disease could have severe effects on free-ranging deer populations, depending on hunting policies and environmental persistence (8,9). CWD has been associated with large decreases in free-ranging mule deer populations in an area of high CWD prevalence (Boulder, Colorado, USA) (5).
PLEASE STUDY THIS MAP, COMPARE FARMED CWD TO WILD CWD...TSS
http://wwwnc.cdc.gov/eid/article/18/3/11-0685-f1.htm
Saturday, February 18, 2012
Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease
CDC Volume 18, Number 3—March 2012
CWD has been identified in free-ranging cervids in 15 US states and 2 Canadian provinces and in ≈100 captive herds in 15 states and provinces and in South Korea (Figure 1, panel B).
http://wwwnc.cdc.gov/eid/article/18/3/11-0685_article.htm
Thursday, February 09, 2012
50 GAME FARMS IN USA INFECTED WITH CHRONIC WASTING DISEASE
http://chronic-wasting-disease.blogspot.com/2012/02/50-game-farms-to-date-in-usa-infected.html
Monday, June 11, 2012
OHIO Captive deer escapees and non-reporting
http://chronic-wasting-disease.blogspot.com/2012/06/ohio-captive-deer-escapees-and-non.html
Tuesday, June 19, 2012
Experimental Oral Transmission of Chronic Wasting Disease to Reindeer (Rangifer tarandus tarandus)
http://chronic-wasting-disease.blogspot.com/2012/06/experimental-oral-transmission-of.html
Monday, June 18, 2012
natural cases of CWD in eight Sika deer (Cervus nippon) and five Sika/red deer crossbreeds captive Korea and Experimental oral transmission to red deer (Cervus elaphus elaphus)
http://chronic-wasting-disease.blogspot.com/2012/06/natural-cases-of-cwd-in-eight-sika-deer.html
Tuesday, July 10, 2012
Chronic Wasting Disease Detected in Far West Texas
http://chronic-wasting-disease.blogspot.com/2012/07/chronic-wasting-disease-detected-in-far.html
SEE FULL HISTORY OF MY EFFORTS TO WARN TAHC ON CWD IN WEST TEXAS SINCE 2001
Monday, March 26, 2012
Texas Prepares for Chronic Wasting Disease CWD Possibility in Far West Texas
http://chronic-wasting-disease.blogspot.com/2012/03/texas-prepares-for-chronic-wasting.html
Thursday, July 12, 2012
CWD aka MAD DEER, ELK DISEASE TEXAS HOUSTON CHRONICLE
Wednesday, July 11, 2012 Brain-eating disease found in Texas deer
http://chronic-wasting-disease.blogspot.com/2012/07/cwd-aka-mad-deer-elk-disease-texas.html
LANCET INFECTIOUS DISEASE JOURNAL
Volume 3, Number 8 01 August 2003
Newsdesk
Tracking spongiform encephalopathies in North America
Xavier Bosch
My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem.
49-year-old Singeltary is one of a number of people who have remained largely unsatisfied after being told that a close relative died from a rapidly progressive dementia compatible with spontaneous Creutzfeldt-Jakob disease (CJD). So he decided to gather hundreds of documents on transmissible spongiform encephalopathies (TSE) and realised that if Britons could get variant CJD from bovine spongiform encephalopathy (BSE), Americans might get a similar disorder from chronic wasting disease (CWD)the relative of mad cow disease seen among deer and elk in the USA. Although his feverish search did not lead him to the smoking gun linking CWD to a similar disease in North American people, it did uncover a largely disappointing situation.
Singeltary was greatly demoralised at the few attempts to monitor the occurrence of CJD and CWD in the USA. Only a few states have made CJD reportable. Human and animal TSEs should be reportable nationwide and internationally, he complained in a letter to the Journal of the American Medical Association (JAMA 2003; 285: 733). I hope that the CDC does not continue to expect us to still believe that the 85% plus of all CJD cases which are sporadic are all spontaneous, without route or source.
Until recently, CWD was thought to be confined to the wild in a small region in Colorado. But since early 2002, it has been reported in other areas, including Wisconsin, South Dakota, and the Canadian province of Saskatchewan. Indeed, the occurrence of CWD in states that were not endemic previously increased concern about a widespread outbreak and possible transmission to people and cattle.
To date, experimental studies have proven that the CWD agent can be transmitted to cattle by intracerebral inoculation and that it can cross the mucous membranes of the digestive tract to initiate infection in lymphoid tissue before invasion of the central nervous system. Yet the plausibility of CWD spreading to people has remained elusive.
Getting data on TSEs in the USA from the government is like pulling teeth, Singeltary argues. You get it when they want you to have it, and only what they want you to have.
SNIP...FULL TEXT ;
http://www.thelancet.com/journals/laninf/article/PIIS1473309903007151/%20fulltext
http://chronic-wasting-disease.blogspot.com/
Wednesday, June 27, 2012
First US BSE Case Since 2006 Underscores Need for Vigilance
Neurology Today 21 June 2012
http://transmissiblespongiformencephalopathy.blogspot.com/2012/06/first-us-bse-case-since-2006.html
Sunday, May 6, 2012
Bovine Spongiform Encephalopathy Mad Cow Disease, BSE May 2, 2012 IOWA State University OIE
http://transmissiblespongiformencephalopathy.blogspot.com/2012/05/bovine-spongiform-encephalopathy-mad.html
http://bseusa.blogspot.com/
http://bse-atypical.blogspot.com/
Thursday, March 29, 2012
atypical Nor-98 Scrapie has spread from coast to coast in the USA 2012
NIAA Annual Conference April 11-14, 2011San Antonio, Texas
http://nor-98.blogspot.com/2012/03/atypical-nor-98-scrapie-has-spread-from.html
Monday, June 11, 2012
another atypical Nor-98 Scrapie case documented in Canada for 2012
http://nor-98.blogspot.com/2012/06/another-atypical-nor-98-scrapie-case.html
Subject: USA BSE GBR ANNEX CONCLUSION
5. CONCLUSION ON THE GEOGRAPHICAL BSE-RISK
5.1 The current GBR as function of the past stability and challenge
• The current geographical BSE risk (GBR) level is III, i.e. it is likely but not
confirmed that domestic cattle are (clinically or pre-clinically) infected with the
BSE-agent.
Note1: It is also worth noting that the current GBR conclusions are not dependent on
the large exchange of imports between USA and Canada. External challenge due to
exports to the USA from European countries varied from moderate to high. These
Annex to the EFSA Scientific Report (2004) 3, 1-17 on the Assessment of the
Geographical BSE Risk of USA
- 16 -
challenges indicate that it was likely that BSE infectivity was introduced into the
North American continent.
Note2: This assessment deviates from the previous assessment (SSC opinion, 2000)
because at that time several exporting countries were not considered a potential risk.
5.2 The expected development of the GBR as a function of the past
and present stability and challenge
• As long as there are no significant changes in rendering or feeding, the stability
remains extremely/very unstable. Thus, the probability of cattle to be (preclinically
or clinically) infected with the BSE-agent persistently increases.
• Since recent improvements in the safety of MBM production in many countries
or significant recent reductions in the incidence of BSE are not taken into
account for the assessment of the external challenge, the external challenge
assessed after 2001 could be overestimated and is the worst case assumption.
However all current GBR conclusions are not dependent on these assumptions
in any of the countries assessed. For future assessments and when the impact of
the production, surveillance and true incidence changes have been fully
quantified, these developments should be taken into account.
http://www.efsa.europa.eu/en/scdocs/doc/3rax1.pdf
When the OIE and the USDA et al collaborated to make legal the trading of Transmissible Spongiform Encephalopathy, when they did away with the BSE GBR risk assessments, where the USA, Canada, and Mexico were categorized as BSE GBR III. please see ;
EFSA concludes that the current GBR level of USA is III, i.e. it is likely but not confirmed that domestic cattle are (clinically or pre-clinically) infected with the BSE-agent. As long as there are no significant changes in rendering or feeding, the stability remains extremely/very unstable. Thus, the probability of cattle to be (pre-clinically or clinically) infected with the BSE-agent persistently increases.
snip...
Spongiform Encephalopathy (BSE) Risk (GBR) were asked by the European Commission (EC) to provide an up-to-date scientific report on the GBR in the United States of America, i.e. the likelihood of the presence of one or more cattle being infected with BSE, pre-clinically as well as clinically, in USA. This scientific report addresses the GBR of USA as assessed in 2004 based on data covering the period 1980-2003. The BSE agent was probably imported into USA and could have reached domestic cattle in the middle of the eighties. These cattle imported in the mid eighties could have been rendered in the late eighties and therefore led to an internal challenge in the early nineties. It is possible that imported meat and bone meal (MBM) into the USA reached domestic cattle and leads to an internal challenge in the early nineties. A processing risk developed in the late 80s/early 90s when cattle imports from BSE risk countries were slaughtered or died and were processed (partly) into feed, together with some imports of MBM. This risk continued to exist, and grew significantly in the mid 90’s when domestic cattle, infected by imported MBM, reached processing. Given the low stability of the system, the risk increased over the years with continued imports of cattle and MBM from BSE risk countries.
EFSA concludes that the current GBR level of USA is III, i.e. it is likely but not confirmed that domestic cattle are (clinically or pre-clinically) infected with the BSE-agent. As long as there are no significant changes in rendering or feeding, the stability remains extremely/very unstable. Thus, the probability of cattle to be (pre-clinically or clinically) infected with the BSE-agent persistently increases. Key words: BSE, geographical risk assessment, GBR, USA, third countries
http://www.efsa.europa.eu/EFSA/efsa_locale-1178620753812_1211902594180.htm
http://www.efsa.europa.eu/en/efsajournal/doc/3r.pdf
5. CONCLUSION ON THE GEOGRAPHICAL BSE-RISK
5.1 The current GBR as function of the past stability and challenge
• The current geographical BSE risk (GBR) level is III, i.e. it is likely but not confirmed that domestic cattle are (clinically or pre-clinically) infected with the BSE-agent.
Note1: It is also worth noting that the current GBR conclusions are not dependent on the large exchange of imports between USA and Canada. External challenge due to exports to the USA from European countries varied from moderate to high. These Annex to the EFSA Scientific Report (2004) 3, 1-17 on the Assessment of the Geographical BSE Risk of USA
- 16 -
challenges indicate that it was likely that BSE infectivity was introduced into the North American continent.
Note2: This assessment deviates from the previous assessment (SSC opinion, 2000) because at that time several exporting countries were not considered a potential risk. 5.2 The expected development of the GBR as a function of the past and present stability and challenge
• As long as there are no significant changes in rendering or feeding, the stability remains extremely/very unstable. Thus, the probability of cattle to be (preclinically or clinically) infected with the BSE-agent persistently increases.
• Since recent improvements in the safety of MBM production in many countries or significant recent reductions in the incidence of BSE are not taken into account for the assessment of the external challenge, the external challenge assessed after 2001 could be overestimated and is the worst case assumption. However all current GBR conclusions are not dependent on these assumptions in any of the countries assessed. For future assessments and when the impact of the production, surveillance and true incidence changes have been fully quantified, these developments should be taken into account.
http://www.efsa.europa.eu/en/scdocs/doc/3rax1.pdf
Annex to the EFSA Scientific Report (2004) 3, 1-17 on the Assessment of the Geographical BSE Risk of USA
please see full text ;
http://www.efsa.europa.eu/en/scdocs/doc/3rax1.pdf
YET, in 2010, tons and tons of banned mad cow protein are still in commerce here in the USA, scientific studies are being misconstrued and manipulated by ARS USDA, which are still going by TSE science that is decades old, while refusing to acknowledge new scientific studies, and FOIA requests are still being held up by the USDA et al on these urgent matters (see source related materials below). CJD of unknown phenotype, in victims that are getting younger, with longer clinical course from first onset of symptoms to death are occurring, in fact, sporadic CJD is still rising, where the TSEs in the different species are mutating here in the USA, and we still have this same dog and pony show by the OIE and USDA et al. IF you go back and look at the Countries that went by these OIE BSE guidelines, most all came down with BSE. I have said it before, I was say it again now, OIE should hang up there jock strap now, since it appears they will buckle every time a country makes some political hay about trade protocol, commodities and futures. IF they are not going to be science based, they should do everyone a favor and dissolve there organization. ...TSS
see full text and reasons why here ;
http://usdameatexport.blogspot.com/2010/06/us-denied-upgraded-bse-status-from-oie.html
http://www.agweekly.com/articles/2010/06/30/commodities/livestock/lvstk10.txt
Geographical BSE risk assessment and its impact on disease detection and dissemination
Original Research Article
Preventive Veterinary Medicine, Available online 1 February 2012,
Mo Salman, Vittorio Silano, Dagmar Heim, Joachim Kreysa
Preventive Veterinary Medicine
1 February 2012
Geographical BSE risk assessment and its impact on disease detection and dissemination
Salman M, Silano V, Heim D, Kreysa J.
Source
Campus Stop 1644, Animal Population Health Institute, College of Veterinary Medicine and Biomedical Sciences, Colorado State University, Fort Collins, CO 80523-1644, USA.
Abstract
Bovine Spongiform Encephalopathy (BSE) rapidly evolved into an issue of major public concern particularly when, in 1996, evidence was provided that this disease had crossed the species barrier and infected humans in the UK with what has become known as "variant Creutzfeldt Jakob Disease" (vCJD). The aim of this paper is to describe the European Geographical BSE risk assessment (GBR) that was successfully used for assessing the qualitative likelihood that BSE could be present in a country where it was not yet officially recognized. It also discusses how this can lead to risk-based and therefore preventive management of BSE at national and international levels. The basic assumption of the GBR method is that the BSE agent is initially introduced into a country's domestic cattle production system through the importation of contaminated feedstuffs or live cattle. This is referred to as an "external challenge". The ability of the system to cope with such a challenge is, in turn, referred to as its "stability": a stable system will not allow the BSE agent to propagate and amplify following its introduction, while an unstable system will. The BSE-status of a country assessed by this system was used by the European Commission as the basis for trade legislation rules for cattle and their products. The GBR was an invaluable tool in evaluating the potential global spread of BSE as it demonstrated how a disease could be transferred through international trade. This was shown to be a critical factor to address in reducing the spread and amplification of BSE throughout the world. Furthermore, GBR resulted in the implementation of additional measures and management activities both to improve surveillance and to prevent transmission within the cattle population.
Copyright © 2012 Elsevier B.V. All rights reserved.
http://www.sciencedirect.com/science/article/pii/S0167587712000244
see more here ;
http://transmissiblespongiformencephalopathy.blogspot.com/2012/02/bovine-spongiform-encephalopathy-bse-31.html
USDA INC. BSE surveillance
this also was a complete failure as well, to a point that the GAO caught the USDA et al in their BSE testing surveillance program, red handed testing cattle they knew were healthy, free of BSE. yes, up to 100 farms testing for mad cow disease, but the animals in question, were all healthy animal brains, and they knew it. but that was not the only failures in the BSE testing program, that was just part of it. the USDA covered up two mad cows in Texas, one finally confirmed after an act of Congress by the OIG made the USDA retest that cow, some 7 months later, and finally confirm, what they already knew with a SECRET test that had tested positive 7 months previously, and finally were forced to confirm this second mad cow in Texas. it got so bad around 2005, that the top prion scientist at the NIH Paul Brown, said "Everything they did on the Texas cow makes everything they did before 2005 suspect," Brown said.
http://www.upi.com/Health_News/2006/03/15/Analysis-What-that-mad-cow-means/UPI-12841142465253/
http://madcowtesting.blogspot.com/2008/08/creekstone-vs-usda-court-of-appeals.html
Wednesday, May 2, 2012
ARS FLIP FLOPS ON SRM REMOVAL FOR ATYPICAL L-TYPE BASE BSE RISK HUMAN AND ANIMAL HEALTH
http://transmissiblespongiformencephalopathy
Saturday, May 26, 2012
Are USDA assurances on mad cow case 'gross oversimplification'?
SNIP...
What irks many scientists is the USDA’s April 25 statement that the rare disease is “not generally associated with an animal consuming infected feed.”
The USDA’s conclusion is a “gross oversimplification,” said Dr. Paul Brown, one of the world’s experts on this type of disease who retired recently from the National Institutes of Health. "(The agency) has no foundation on which to base that statement.”
“We can’t say it’s not feed related,” agreed Dr. Linda Detwiler, an official with the USDA during the Clinton Administration now at Mississippi State.
In the May 1 email to me, USDA’s Cole backed off a bit. “No one knows the origins of atypical cases of BSE,” she said
The argument about feed is critical because if feed is the cause, not a spontaneous mutation, the California cow could be part of a larger outbreak.
SNIP...
http://bseusa.blogspot.com/2012/05/are-usda-assurances-on-mad-cow-case.html
P.9.21
Molecular characterization of BSE in Canada
Jianmin Yang1, Sandor Dudas2, Catherine Graham2, Markus Czub3, Tim McAllister1, Stefanie Czub1 1Agriculture and Agri-Food Canada Research Centre, Canada; 2National and OIE BSE Reference Laboratory, Canada; 3University of Calgary, Canada
Background: Three BSE types (classical and two atypical) have been identified on the basis of molecular characteristics of the misfolded protein associated with the disease. To date, each of these three types have been detected in Canadian cattle.
Objectives: This study was conducted to further characterize the 16 Canadian BSE cases based on the biochemical properties of there associated PrPres. Methods: Immuno-reactivity, molecular weight, glycoform profiles and relative proteinase K sensitivity of the PrPres from each of the 16 confirmed Canadian BSE cases was determined using modified Western blot analysis.
Results: Fourteen of the 16 Canadian BSE cases were C type, 1 was H type and 1 was L type. The Canadian H and L-type BSE cases exhibited size shifts and changes in glycosylation similar to other atypical BSE cases. PK digestion under mild and stringent conditions revealed a reduced protease resistance of the atypical cases compared to the C-type cases. N terminal- specific antibodies bound to PrPres from H type but not from C or L type. The C-terminal-specific antibodies resulted in a shift in the glycoform profile and detected a fourth band in the Canadian H-type BSE.
Discussion: The C, L and H type BSE cases in Canada exhibit molecular characteristics similar to those described for classical and atypical BSE cases from Europe and Japan. This supports the theory that the importation of BSE contaminated feedstuff is the source of C-type BSE in Canada.
*** It also suggests a similar cause or source for atypical BSE in these countries.
http://www.prion2009.com/sites/default/files/Prion2009_Book_of_Abstracts.pdf
Thursday, August 12, 2010
Seven main threats for the future linked to prions
First threat
The TSE road map defining the evolution of European policy for protection against prion diseases is based on a certain numbers of hypotheses some of which may turn out to be erroneous. In particular, a form of BSE (called atypical Bovine Spongiform Encephalopathy), recently identified by systematic testing in aged cattle without clinical signs, may be the origin of classical BSE and thus potentially constitute a reservoir, which may be impossible to eradicate if a sporadic origin is confirmed.
***Also, a link is suspected between atypical BSE and some apparently sporadic cases of Creutzfeldt-Jakob disease in humans. These atypical BSE cases constitute an unforeseen first threat that could sharply modify the European approach to prion diseases.
Second threat
snip...
http://www.neuroprion.org/en/np-neuroprion.html
in the url that follows, I have posted
SRM breaches first, as late as 2011.
then
MAD COW FEED BAN BREACHES AND TONNAGES OF MAD COW FEED IN COMMERCE up until 2007, when they ceased posting them.
then,
MAD COW SURVEILLANCE BREACHES.
Friday, May 18, 2012
Update from APHIS Regarding a Detection of Bovine Spongiform Encephalopathy (BSE) in the United
States Friday May 18, 2012
http://transmissiblespongiformencephalopathy.blogspot.com/2012/05/update-from-aphis-regarding-detection.html
Thursday, June 21, 2012
MEATINGPLACE.COM WAVES MAGIC WAND AND EXPECTS THE USDA MAD COW FOLLIES BSE TO BE GONE
http://bse-atypical.blogspot.com/2012/06/meatingplacecom-waves-magic-wand-and.html
Thursday, June 14, 2012
R-CALF USA Calls USDA Dishonest and Corrupt; Submits Fourth Request for Extension
R-CALF United Stockgrowers of America
http://madcowusda.blogspot.com/2012/06/r-calf-usa-calls-usda-dishonest-and.html
Friday, May 25, 2012
R-CALF USDA’s New BSE Rule Eliminates Important Protections Needed to Prevent BSE Spread
http://bseusa.blogspot.com/2012/05/r-calf-usdas-new-bse-rule-eliminates.html
Monday, June 18, 2012
R-CALF Submits Incomplete Comments Under Protest in Bizarre Rulemaking “Bovine Spongiform Encephalopathy; Importation of Bovines and Bovine Products”
http://madcowusda.blogspot.com/2012/06/r-calf-submits-incomplete-comments.html
Saturday, March 5, 2011
MAD COW ATYPICAL CJD PRION TSE CASES WITH CLASSIFICATIONS PENDING ON THE RISE IN NORTH AMERICA
http://transmissiblespongiformencephalopathy.blogspot.com/2011/03/mad-cow-atypical-cjd-prion-tse-cases.html
Wednesday, November 09, 2011
Case report Sporadic fatal insomnia in a young woman: A diagnostic challenge: Case Report TEXAS
HOW TO TURN A POTENTIAL MAD COW VICTIM IN THE USA, INTO A HAPPENSTANCE OF BAD LUCK, A SPONTANEOUS MUTATION FROM NOTHING. OR WAS IT $$$
http://creutzfeldt-jakob-disease.blogspot.com/2011/11/case-report-sporadic-fatal-insomnia-in.html
Sunday, February 12, 2012
National Prion Disease Pathology Surveillance Center Cases Examined1 (August 19, 2011) including Texas
snip...
CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER
Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas
Irma Linda Andablo CJD Victim, she died at 38 years old on February 6, 2010 in Mesquite Texas. She left 6 Kids and a Husband. The Purpose of this web is to give information in Spanish to the Hispanic community, and to all the community who want's information about this terrible disease.-
Physician Discharge Summary, Parkland Hospital, Dallas Texas
Admit Date: 12/29/2009 Discharge Date: 1/20/2010 Attending Provider: Greenberg, Benjamin
Morris; General Neurology Team: General Neurology Team
snip...
The husband says that they have lived in Nebraska for the past 21 years. They had seen a doctor there during the summer time who prescribed her Seroquel and Lexapro, Thinking these were sx of a mood disorder. However, the medications did not help and she continued to deteriorate clinically. Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. The husband says that he does not know any fellow workers with a similar illness. He also says that she did not have any preceeding illness or travel.
snip...
http://www.recordandoalinda.com/index.php?option=com_content&view=article&id=19:cjd-english-info&catid=9:cjd-ingles&Itemid=8
>>> Up until about 6 years ago, the pt worked at Tyson foods where she worked on the assembly line, slaughtering cattle and preparing them for packaging. She was exposed to brain and spinal cord matter when she would euthanize the cattle. <<<
SEE MORE HERE ;
CJD TEXAS 38 YEAR OLD FEMALE WORKED SLAUGHTERING CATTLE EXPOSED TO BRAIN AND SPINAL CORD MATTER
http://cjdtexas.blogspot.com/2010/03/cjd-texas-38-year-old-female-worked.html
Sunday, February 12, 2012
National Prion Disease Pathology Surveillance Center Cases Examined1 (August 19, 2011) including Texas
http://transmissiblespongiformencephalopathy.blogspot.com/2012/02/national-prion-disease-pathology.html
Tuesday, November 08, 2011
Can Mortality Data Provide Reliable Indicators for Creutzfeldt-Jakob Disease Surveillance? A Study in France from 2000 to 2008 Vol. 37, No. 3-4, 2011
Original Paper
Conclusions:These findings raise doubt about the possibility of a reliable CJD surveillance only based on mortality data.
http://creutzfeldt-jakob-disease.blogspot.com/2011/11/can-mortality-data-provide-reliable.html
price of prion poker goes up again $$$
Monday, June 11, 2012
Guidance for Industry Draft Guidance for Industry: Amendment to “Guidance for Industry: Revised Preventive Measures to Reduce the Possible Risk of Transmission of Creutzfeldt-Jakob Disease and Variant Creutzfeldt-Jakob Disease by Blood and Blood Products”
http://creutzfeldt-jakob-disease.blogspot.com/2012/06/guidance-for-industry-draft-guidance.html
Sunday, June 3, 2012
A new neurological disease in primates inoculated with prion-infected blood or blood components
http://transmissiblespongiformencephalopathy.blogspot.com/2012/06/new-neurological-disease-in-primates.html
PLEASE REMEMBER ;
The Akron, Ohio-based CJD Foundation said the Center for Disease Control revised that number in October of 2004 to about one in 9,000 CJD cases per year in the population group age 55 and older.
HAVE YOU GOT YOUR CJD QUESTIONNAIRE ASKING REAL QUESTIONS PERTAINING TO ROUTE AND SOURCE OF THE TSE AGENT THAT KILLED YOUR LOVED ONE ???
if not, why not...
Friday, November 30, 2007
CJD QUESTIONNAIRE USA CWRU AND CJD FOUNDATION
http://cjdquestionnaire.blogspot.com/2007/11/cjd-questionnaire.html
http://cjdquestionnaire.blogspot.com/
Terry S. Singeltary Sr. on the Creutzfeldt-Jakob Disease Public Health Crisis
http://www.youtube.com/watch?v=zf3lfz9NrT4
http://www.youtube.com/watch?v=c0tWkNvhO4g
http://www.youtube.com/watch?v=zf3lfz9NrT4&feature=results_main&playnext=1&list=PL780BE2AF0B62A944
full text with source references ;
http://transmissiblespongiformencephalopathy.blogspot.com/2011/08/terry-singeltary-sr-on-creutzfeldt.html
Sunday, August 21, 2011
The British disease, or a disease gone global, The TSE Prion Disease (SEE VIDEO)
http://transmissiblespongiformencephalopathy.blogspot.com/2011/08/british-disease-or-disease-gone-global.html
U.S.A. HIDING MAD COW DISEASE VICTIMS AS SPORADIC CJD ? (see video at bottom)
http://creutzfeldt-jakob-disease.blogspot.com/2009/07/usa-hiding-mad-cow-disease-victims-as.html
WHO WILL FOLLOW THE CHILDREN FOR CJD SYMPTOMS ???
Saturday, May 2, 2009
U.S. GOVERNMENT SUES WESTLAND/HALLMARK MEAT OVER USDA CERTIFIED DEADSTOCK DOWNER COW SCHOOL LUNCH PROGRAM
http://downercattle.blogspot.com/2009/05/us-government-sues-westlandhallmark.html
OUR SCHOOL CHILDREN ALL ACROSS THE USA WERE FED THE MOST HIGH RISK CATTLE FOR MAD COW DISEASE FOR 4 YEARS I.E. DEAD STOCK DOWNER CATTLE VIA THE USDA AND THE NSLP.
WHO WILL WATCH OUR CHILDREN FOR THE NEXT 5+ DECADES ???
DID YOUR CHILD CONSUME SOME OF THESE DEAD STOCK DOWNER COWS, THE MOST HIGH RISK FOR MAD COW DISEASE ???
you can check and see here ;
http://www.fns.usda.gov/fns/safety/pdf/Hallmark-Westland_byState.pdf
http://downercattle.blogspot.com/
Tuesday, June 26, 2012
Creutzfeldt Jakob Disease Human TSE report update North America, Canada, Mexico, and USDA PRION UNIT as of May 18, 2012
type determination pending Creutzfeldt Jakob Disease (tdpCJD), is on the rise in Canada and the USA
http://creutzfeldt-jakob-disease.blogspot.com/2012/06/creutzfeldt-jakob-disease-human-tse.html
Wednesday, May 16, 2012
Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ?
Proposal ID: 29403
http://betaamyloidcjd.blogspot.com/2012/05/alzheimers-disease-and-transmissible.html
layperson
Terry S. Singeltary Sr.
P.O. Box 42
Bacliff, Texas USA 77518
flounder9@verizon.net
http://nor-98.blogspot.com/
http://creutzfeldt-jakob-disease.blogspot.com/
http://transmissiblespongiformencephalopathy.blogspot.com/
Tuesday, July 29, 2008
Heidenhain Variant Creutzfeldt Jakob Disease Case Report
FINAL AUTOPSY DIAGNOSIS
I. Brain: Creutzfeldt-Jakob disease, Heidenhain variant.
SKROLL down a bit for Mom's autopsy of hvCJD. ...
http://creutzfeldt-jakob-disease.blogspot.com/2008/07/heidenhain-variant-creutzfeldt-jakob.html
Wednesday, May 16, 2012
OIE UPDATE BOVINE SPONGIFORM ENCEPHALOPATHY UNITED STATES OF AMERICA MAY 15, 2012
http://transmissiblespongiformencephalopathy.blogspot.com/2012/05/oie-update-bovine-spongiform.html
RESPONSE TO PUBLIC COMMENTS
of Bovine Spongiform Encephalopathy Update, October 31, 2005 INTRODUCTION The United States Department of Agriculture’s Food Safety and Inspection Service (FSIS) held a public meeting on July 25, 2006 in Washington, D.C. to present findings from the Harvard Risk Assessment of Bovine Spongiform Encephalopathy Update, October 31, 2005 (report and model located on the FSIS website:
http://www.fsis.usda.gov/Science/Risk_Assessments/index.asp).
Comments on technical aspects of the risk assessment were then submitted to FSIS. Comments were received from Food and Water Watch, Food Animal Concerns Trust (FACT), Farm Sanctuary, R-CALF USA, Linda A Detwiler, and Terry S. Singeltary. This document provides itemized replies to the public comments received on the 2005 updated Harvard BSE risk assessment. Please bear the following points in mind:
http://www.fsis.usda.gov/PDF/BSE_Risk_Assess_Response_Public_Comments.pdf
IN SHORT, AND IN A NUT SHELL ;
(Adopted by the International Committee of the OIE on 23 May 2006)
11. Information published by the OIE is derived from appropriate declarations made by the official Veterinary Services of Member Countries. The OIE is not responsible for inaccurate publication of country disease status based on inaccurate information or changes in epidemiological status or other significant events that were not promptly reported to the Central Bureau,
http://www.oie.int/eng/Session2007/RF2006.pdf
with sad regards,
terry
